Welcome to Mother Earth Living, the authority on green lifestyle and design. Each issue of Mother Earth Living features advice to create naturally healthy and nontoxic homes for yourself and your loved ones. With Mother Earth Living by your side, you’ll discover all the best and latest information you want on choosing natural remedies and practicing preventive medicine; cooking with a nutritious and whole-food focus; creating a nontoxic home; and gardening for food, wellness and enjoyment. Subscribe to Mother Earth Living today to get inspired on the art of living wisely and living well.
Thyroid hormone is critical for normal brain development in babies. Infants requiring thyroid hormone therapy should NOT be treated with purchased liquid suspensions, since the active hormone may deteriorate once dissolved and the baby could receive less thyroid hormone than necessary. Instead, infants with hypothyroidism should receive their thyroid hormone by crushing a single tablet daily of the correct dose and suspending it in one teaspoon of liquid and administering it properly.
Think milk, butter, cheese, and meat. If you buy the cheap, conventionally raised versions at the supermarket, those types of deliciousness can also disrupt all your thyroid’s hard work. You omnivores (like us) can avoid this dilemma by choosing organic, or at least antibiotic-free and hormone-free meats and dairy. It’ll save you in the end, with fewer medical costs down the line.
To document that this was a result of trends toward lower doses, an unblinded study tracked well-being according to various doses and found that the highest well-being was achieved at supraoptimal doses, resulting in a suppressed TSH (65). However, a blinded trial did not reproduce this finding (66). In a call to the public, a 1997 British Thyroid Foundation newsletter asked readers to recount personal history of residual hypothyroid symptoms. More than 200 patients responded, 54 of whom specifically mentioned that they did not feel well despite normal serum markers of thyroid function (67, 68). Because of this surge in symptomatic patients, some clinicians advocated titrating dose by symptoms rather than serum TSH, reminiscent of the period before the 1970s (69).
The vast majority of individuals—one in seven are women—with hypothyroidism in the US have Hashimoto’s thyroiditis, an autoimmune disease in which your body doesn’t produce enough thyroid hormone, but this isn’t caused by iodine levels in the diet.2 Other less common causes of hypothyroidism include a deficiency of iodine in the diet, taking certain medications that interfere with thyroid absorption, surgical removal of the thyroid, and a genetic disorder.
As for what’s causing your condition, this of course can vary. Many times it is caused by lifestyle factors, such as poor eating habits, lack of sleep, not handling stress well, etc. Other times it can be environmental toxins or an infection causing or contributing to such a condition. Genetics can also be a factor, although research shows that lifestyle and environmental factors play a much greater role in the development of these conditions. In fact, many people with a genetic marker for hypothyroidism or Hashimoto’s Thyroiditis can be helped a great deal by modifying some of their lifestyle factors, which is great news.
In humans, a factor associated with response to combination therapy in a large clinical trial is the Thr92Ala polymorphism in the type 2 deiodinase gene (DIO2), wherein the subpopulation of patients with this genetic alteration had improved well-being and preference for combination therapy (7). This has led investigators to consider whether this polymorphism could confer a defect in the D2 pathway, but normal Thr92AlaD2 enzyme kinetics have been demonstrated (73). Only recently has the Thr92AlaD2 protein been found to have a longer half-life, ectopically localize in the Golgi apparatus, and significantly alter the genetic fingerprint in cultured cells and in the temporal pole of the human brain without evidence of reduced thyroid hormone signaling (74). The significance of these studies transcends the thyroid field—this polymorphism has now been associated with a constellation of diseases, including mental retardation, bipolar disorder, and low IQ (75). If hypothyroid carriers of Thr92AlaD2 benefit from alternate therapeutic strategies in replicate studies, then personalized medicine—based on genotype— may have a role.
Selenium: Selenium is one the MOST IMPORTANT when it comes to healthy thyroid function! It is incorporated into key enzymes involved in several metabolic pathways implicated in thyroid hormone metabolism; additionally, it plays an antioxidant role in the regulation of the immune system. There are strong links between selenium deficiencies and auto-immune thyroid problems (48, 49).
Hypothyroidism is a secondary cause of dyslipidemia, typically manifesting in elevation of low-density lipoprotein and total cholesterol levels. It is clear that treatment resulting in the normalization of the serum TSH is associated with reduction in total cholesterol levels (54), but whether total cholesterol is fully normalized by l-thyroxine monotherapy is less well-defined. An analysis of 18 studies on the effect of thyroid hormone replacement on total cholesterol levels in overt hypothyroidism showed a reduction in the total cholesterol level in all 18 studies; however, in 14 of the 18 studies, the mean post treatment total cholesterol level remained above the normal range (>200 mg/dL [>5.18 mmol/L]) (55). These findings suggest that lipid measures are not fully restored despite normalization of the serum TSH (56). Whether the degree of dyslipidemia remaining in l-thyroxine-treated patients with a normal TSH is clinically significant is unknown, given that the benefit of thyroid hormone replacement in subclinical hypothyroidism is itself controversial (57, 58).
Iodine intake often isn’t readily apparent on a dietary recall since the amount in foods is largely dependent on levels in the soil and added salt. However, Schneider says, “Clients taking iodine tablets are a red flag. Frequent intake of foods such as seaweed or an avoidance of all iodized salt may serve as signs that further exploration is needed.”
The best diet for your thyroid requires more than just iodine, selenium, and vitamin D, says Ilic. And—perhaps unsurprisingly—foods that are high in antioxidants are also good for your thyroid. One 2008 study by researchers from Turkey suggests that people with hypothyroidism have higher levels of harmful free radicals than those without the condition.
Trisha Gilkerson is a homeschooling mom to four crazy boys. She blogs with her awesome hubby Luke at Intoxicated on Life where they talk about faith, homeschooling, and health. They’ve authored the Write Through the Bible curriculum and family Bible Studies and have recently released their first healthy living book – Weeding Out Wheat: A Simple Faith Based Guide. They love connecting with their readers, so be sure to follow them on their blog, Facebook, Twitter, Google+, and Pinterest.
Other causes of hypothyroidism include surgical removal of the thyroid (usually for cancer), radiation therapy of the head and neck, or complications of medical therapies for hyperthyroidism. (Patients with overactive thyroids are often treated with radioactive iodine or anti-thyroid medications that reduce thyroid functioning. These effects can be extensive and permanent, and thyroid supplementation is often required flowing these interventions.) Certain medications can worsen or promote hypothyroidism or interfere with thyroid replacement therapy. One such drug is lithium, used for treating psychiatric conditions such as bipolar disorder.
Soy for thyroid health is controversial: There's some research that suggests soy might negatively affect your thyroid gland under certain circumstances, like if you have an iodine deficiency. (Something to keep in mind: A 2011 study of vegetarians and vegans in the Boston area found that some vegans did have a mild iodine deficiency, most likely because they don't eat animal and dairy products). But other research presented at the 2014 Endocrine Society's annual meeting found that unless you have thyroid problems already, soy probably won't have any effect on it. Again, says Ilic, as long as you're eating normal amounts of soy, there's no reason to worry it'll hurt your thyroid.
Emphasizing lean proteins, vegetables, fruits, heart-healthy fats and omega-3s, high-fiber foods, and appropriate portions can help manage or prevent illnesses associated with thyroid disease. As Schneider notes, “It’s eating for prevention of all these diseases that accompany thyroid disease: heart disease, diabetes, cancer, and more.” As an added bonus, fiber can relieve constipation that people with hypothyroidism often experience.
Clara Schneider, MS, RD, RN, CDE, LDN, of Outer Banks Nutrition and author of numerous books, including The Everything Thyroid Diet Book, says, “The No. 1 priority is to get the thyroid disease under control. Clients need to have labs and medications addressed first. Weight changes are just not going to happen before all of that is under control.” She notes that Hashimoto’s typically occurs around menopause, which compounds the weight gain issue that many women experience during that time.
l-Thyroxine monotherapy for athyreotic rats results in a high T4:T3 ratio at doses sufficient to normalize serum TSH levels (8). Yet, the brain, liver, and skeletal muscle tissues of these l-thyroxine–treated animals continue to exhibit markers of hypothyroidism (9), probably because of the inability of l-thyroxine monotherapy to restore tissue levels of T3 (8). This is probably a direct consequence of lower serum T3 levels and the relatively high T4 concentration in these tissues, which inactivates the type 2 iodothyronine deiodinase (D2). In the hypothalamus, loss of D2 is minimal in the presence of T4, which increases sensitivity to T4 levels and explains TSH normalization, despite relatively lower levels of serum T3. Only combination therapy with l-thyroxine plus l-triiodothyronine normalized all thyroid hormone–dependent measures (9), including serum and tissue T3 levels (8). Whether tissue-specific markers of hypothyroidism are restored with l-thyroxine monotherapy in humans remains to be determined, as does the ability of l-thyroxine plus l-triiodothyronine combination therapy to normalize the serum T4:T3 ratio without adverse events. The development of a novel drug delivery system for l-triiodothyronine would facilitate these studies (5).
Think twice before reheating your plastic bowl of takeout soup or keeping that frozen dinner in its original container when you microwave it. Put it on a plate or in a bowl made from ceramics like bone china, stoneware, porcelain or glazed earthenware. Your thyroid is part of your endocrine system, and you can disrupt it by heating food in plastic. The National Institute of Environmental Health Sciences says endocrine disruptors are in many everyday plastic products, including bottles, food, and containers with BPA. Endocrine disruptors work by mimicking naturally occurring hormones in the body, like thyroid hormones.
Gluten intolerance is highly associated with inflammatory disorders of all kinds (63). It is also a contributing factor in many autoimmune diseases such as celiac disease, rheumatoid arthritis, type I diabetes, Hashimoto’s thyroiditis, autoimmune cardiomyopathy, lymphoma and dermatitis herpetiformis (skin disease) among others (64, 65). If you have a thyroid problem or just want to avoid having a future thyroid problem, the first place to start is on a gluten-free nutrition program!
However, iodine intake has dropped during the past few decades. Americans get approximately 70% of their salt intake from processed foods that, in the United States and Canada, generally don’t contain iodine. A 2012 Centers for Disease Control and Prevention report indicates that, on average, Americans are getting adequate amounts of iodine, with the potential exception of women of childbearing age.10
It’s commonly believed that hypothyroidism is due to insufficient iodine, but this isn’t true. Dr. Kharrazian states that if you have Hashimoto’s, taking supplemental iodine is like throwing gasoline on a fire, so eschew iodine supplements and iodized salt. Primary sources of iodine: sea vegetables and seafood. Secondary sources: eggs, asparagus, lima beans, mushrooms, spinach, sesame seeds, summer squash, Swiss chard, and garlic.
Brazil nuts are packed with another nutrient that helps regulate thyroid hormones: selenium. In one 2003 study by researchers in France, women who consumed higher amounts of selenium were less likely to develop goiters and thyroid tissue damage than those who didn't. Plus, it may also help stave off long-term thyroid damage in people with thyroid-related problems like Hashimoto's and Graves' disease, according to a 2013 review in the journal Clinical Endocrinology.
Every three months, you will repeat this process. This repetition is to ensure that you are staying healthy and that the medication is working. As you approach the end of your 90-day prescription, we'll order new lab tests and book a quick check-in with your doctor to make sure that the treatment plan is working for you. If you have any questions in the meantime, you can always give PlushCare a call at 1-888-529-3472, where our Care Coordinators are ready and willing to assist you.
Your thyroid needs iodine to work properly and produce enough TH for your body's needs. Don't get enough iodine, and you run the risk of hypothyroidism or a goiter (a thyroid gland that becomes enlarged to make up for the shortage of thyroid hormone). Most Americans have no problem getting enough iodine, since table salt is iodized—but if you're on a low-sodium diet (as an increasing number of Americans are for their heart health) or follow a vegan diet (more on that later), then you may need to up your intake from other sources.
In the 1995 American Thyroid Association (ATA) guidelines, biological and synthetic thyroid hormone preparations containing T4 plus T3 were not recommended out of concern for fluctuating and often elevated serum T3 concentrations (71). In conjunction with the American Association of Clinical Endocrinologists in 2012, the ATA continued to recommend l-thyroxine monotherapy and noted that evidence does not support using synthetic combination therapies; in addition, they stated that “desiccated thyroid hormone should not be used for the treatment of hypothyroidism” (72). In 2014, the ATA recommendations evolved with the recognition that 1) serum T3 levels might not be normalized in all l-thyroxine–treated hypothyroid patients and 2) some patients remain symptomatic while receiving l-thyroxine monotherapy. Titration of l-thyroxine dose to achieve normal TSH concentrations remains a first-line approach, but trials with combination therapy can be considered. In addition, the guidelines recognize that although superiority data are lacking, some patients do experience a clinical response with desiccated thyroid preparations or combination therapy with l-thyroxine plus l-triiodothyronine (1). The European Thyroid Association has similar recommendations (2).
You want to detox your liver and your gut, as this is where the T4 hormone (inactive hormone) gets converted to T3, the active hormone that actually powers us up. Most of our body cells need T3, not just T4. If you are taking Synthroid, you are taking a synthetic version of T4 that still needs to be converted to T3. If you have a sluggish liver and gut, you won’t convert properly.
Affiliate Disclosure: There are links on this site that can be defined as affiliate links. This means that I may receive a small commission (at no cost to you) if you purchase something when clicking on the links that take you through to a different website. By clicking on the links, you are in no way obligated to buy.
Please Note: The material on this site is provided for informational purposes only and is not medical advice. Always consult your physician before beginning any diet or exercise program.
Copyright © thembcookbook.com