Do a little Googling, and you might turn up a page or two claiming that cruciferous vegetables can cause thyroid troubles. The truth is a little murkier. While it's true that these veggies contain compounds called glucosinolates, which might interfere with your body's production of thyroid hormones in high amounts, it's pretty unlikely that they'll harm your thyroid if you're eating normal-size servings. One case report in the New England Journal of Medicine highlighted the story of an 88-year-old woman who showed up to the ER with hypothyroidism after eating about 2 or 3 pounds of bok choy a day—but, as Ilic points out, "that's not a normal amount."
Hi, Dawn: Yes, there’s definitely a TON of conflicting information out there. When it comes to cruciferous vegetables and the thyroid, it’s all about raw veggies, not cooked. Raw cruciferous veg contains a compound called goitrogens, which might impact thyroid function by impairing thyroid peroxidase, an enzyme normally found in the thyroid gland. I don’t think this means that you should NEVER eat a single serving of raw cruciferous veggies if you have thyroid issues. But just that you shouldn’t overdo it and eat raw daily. Hope that helps!
Dr. Shiel received a Bachelor of Science degree with honors from the University of Notre Dame. There he was involved in research in radiation biology and received the Huisking Scholarship. After graduating from St. Louis University School of Medicine, he completed his Internal Medicine residency and Rheumatology fellowship at the University of California, Irvine. He is board-certified in Internal Medicine and Rheumatology.
“The biggest factors that help with weight loss are calorie- and carbohydrate-controlled meal plans,” says Sheila Dean, DSc, RD, LD, CCN, CDE, of the Palm Harbor Center for Health & Healing in Florida. “Naturally I try to ensure [clients are] eating a whole foods-based, minimally processed diet with at least 2 L of water daily.” Schneider agrees that a heart-healthy eating plan is fundamental. “The diet should emphasize more vegetables, leaner meats, more beans, fiber, and fluids. We need to look at intake of sugars, added fats, fast food, and meals out.”
A clinical trial investigating symptoms found that patients receiving l-thyroxine monotherapy, even with a normal TSH, displayed substantial impairment in psychological well-being compared with controls of similar age and sex (3). Because some hypothesized that this phenomenon came about only after adoption of l-thyroxine monotherapy, a study assessed combination therapy with l-thyroxine and l-triiodothyronine. Remarkably, the latter study showed that psychological measures improve in patients receiving combination therapy until serum TSH level is normal (6). In another study comparing l-thyroxine monotherapy versus desiccated thyroid, in which both groups had a normal TSH, many patients preferred desiccated thyroid and lost weight (60). Unfortunately, the solution to this complex problem is not as simple as reverting to combination therapy; the more than a dozen clinical trials on the subject have not shown benefit of superiority and preference for combination therapy, as previously reviewed (1, 3, 70).
1. Jonklaas J, Bianco AC, Bauer AJ, Burman KD, Cappola AR, Celi FS, et al. American Thyroid Association Task Force on Thyroid Hormone Replacement. Guidelines for the treatment of hypothyroidism: prepared by the American Thyroid Association Task Force on Thyroid Hormone Replacement. Thyroid. 2014;24:1670–1751. [PMID: 25266247] [PMC free article] [PubMed]
Thyroid hormone is critical for normal brain development in babies. Infants requiring thyroid hormone therapy should NOT be treated with purchased liquid suspensions, since the active hormone may deteriorate once dissolved and the baby could receive less thyroid hormone than necessary. Instead, infants with hypothyroidism should receive their thyroid hormone by crushing a single tablet daily of the correct dose and suspending it in one teaspoon of liquid and administering it properly.
From the early 1890s through the mid-1970s, desiccated thyroid was the preferred form of therapy for hypothyroidism (Appendix Table, available at www.annals.org). This preference was reinforced by the unique ability of desiccated thyroid to reproduce a normal serum PBI (33). The predominance of natural thyroid products was illustrated by prescribing patterns in the United States: In 1965, approximately 4 of every 5 prescriptions for thyroid hormone were for natural thyroid preparations (38). Concerns about inconsistencies in the potency of these tablets arose (26) after the discovery that some contained anywhere from double to no detectable metabolic activity (39). The shelf-life of desiccated tablets was limited, especially if the tablets were kept in humid conditions (36). There were reports of patients not responding to desiccated thyroid altogether because their tablets contained no active thyroid hormone. It was not until 1985 that the revision of the U.S. Pharmacopeia standard from iodine content to T3/thyroxine (T4) content resulted in stable potency (38), but by then the reputation of natural thyroid products was tarnished (40).
It is absolutely critical for any physician who is treating someone with a thyroid disorder to test for thyroid antibodies. Unfortunately, few mainstream medical doctors test for thyroid anti-bodies and so most do not ever get the proper diagnosis. In the medical system, an auto-immune condition, a sluggish thyroid, a burned out pituitary gland and a T4-T3 conversion problem are all treated the same way, with synthetic T4 such as synthroid or a T3 medication like levothyroxin.
In the developed world, where protein is plentiful and many countries add iodine to salt and processed foods, we don’t typically need to worry about protein malnutrition or iodine deficiency. However, the rest of the world is not so lucky. More than 2 billion people around the world suffer from hypothyroidism due to iodine deficiency. 2 billion! We are told that the reason for this planetary epidemic is that iodine comes from the ocean, and that the soil of inland areas has had most of its iodine washed away over time by erosion:
The symptoms of hypothyroidism are often subtle. They are not specific (which means they can mimic the symptoms of many other conditions) and are often attributed to aging. People with mild hypothyroidism may have no signs or symptoms. The symptoms generally become more obvious as the condition worsens and the majority of these complaints are related to a metabolic slowing of the body. Common symptoms and signs include:
For instance, soy foods and the broccoli family (broccoli, cabbage, kale, Brussels sprouts, and collard greens) have all been said to cause thyroid dysfunction, but they also have many other health benefits. Research on these foods to date has been less than conclusive. In one study, rats fed high concentrations of soy had problems with their thyroid.
– Gluten. Gluten is compound of glutein and gliadin proteins. Gliadin’s molecular structure is similar to the thyroid gland, so when the inmune system tags it for destruction not only destroys the protein gliadin but also attacks the thyroid tissue affecting the secretion of the thyroid hormone. The gluten from refined flour is much worse than gluten coming from natural sources as whole barley or oats.
One of the most powerful things you can do to help your health and metabolism is to drink enough water. Water helps your metabolism function more efficiently and can help reduce your appetite, get rid of water retention and bloating, improve your digestion and elimination, and combat constipation. Some experts even say that we should drink one ounce of water per pound of scale weight.
55. National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (Adult Treatment Panel III) Third Report of the National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (Adult Treatment Panel III) final report. Circulation. 2002;106:3143–3421. [PMID: 12485966] [PubMed]
Most physicians diagnose hypothyroidism by simple blood tests that measure the level of TSH (thyroid stimulating hormone), which is made by the pituitary gland in response to thyroid hormone and the body’s needs, and indicates thyroid status. As levels of thyroid hormones fall, the pituitary releases TSH to stimulate the thyroid to produce more hormone. Clinicians may also measure circulating levels of T-3 and T-4, which are the thyroid hormones themselves. Low levels of T-4 and high levels of TSH reveal an underactive thyroid. Other variants of hypothyroidism can exist. Patients can have no symptoms and normal serum thyroid hormone levels, but elevated TSH. Others can have symptoms, but normal TSH and T-4 levels. Patients with either of these variants may benefit from supplementation. In addition, someone with a temporary illness might have a completely normal thyroid but high TSH, a condition called “sick euthyroid” which usually resolves without any intervention.
For starters, consider the effect that hypothyroidism can have on weight. Hypothyroidism (also called low thyroid or underactive thyroid) is marked by insufficient hormone production in the thyroid — the butterfly-shaped gland located at the bottom-front of your neck. This gland affects the body’s metabolic processes, and often, sudden weight gain is an early sign of low thyroid.
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